NGWỌTA GLUKOS: NTUZIAKA MAKA IJI NTỤTỤ

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Ọrịa shuga bụ otu n'ime ndị iro kachasị arịa ọrịa shuga. Molemụ irighiri ihe ya, n’agbanyeghi oke ibu n’ihe metụtara irighiri akwara, nwere ike ịhapụ ọdụ ọbara ọbara ngwa ngwa.

Ya mere, site na oghere intercellular, dextrose gafere n’ime sel. Usoro a ghọrọ isi ihe kpatara mmepụta insulin ọzọ.

N'ihi mgbapụta a, metabolism na mmiri na carbon dioxide na-eme. Ọ bụrụ n ’ịba ụba oke dextrose na oke ọbara, mgbe ahụ oke akụrụ ọgwụ a na-enweghị ihe mgbochi gboro akịrị ga-ewepụ.

Ngwakọta ahụ na atụmatụ nke ihe ngwọta ya

Ọgwụ nwere maka 100 ml ọ bụla:

glucose 5 g ma ọ bụ 10 g (ihe na-arụ ọrụ);
sodium chloride, mmiri maka injection 100 ml, hydrochloric acid 0.1 M (ndị mgbe ochie).

Ngwọta glucose bụ mmiri na-enweghị agba ma ọ bụ nke na-acha odo odo.

Ọ bụ glucose bụ monosaccharide dị mkpa nke na-ekpuchi akụkụ nke mmefu ike. Ọ bụ isi mmalite nke carbohydrates na-adị ngwa ngwa. Ihe dị ka caloric nke ihe ahụ bụ 4 kcal kwa gram.

Ihe mejupụtara ọgwụ ahụ nwere ike inwe mmetụta dịgasị iche: welie usoro oxidative na mbelata, melite ọrụ antitoxic nke imeju. Mgbe nchịkwa intraven, ihe ahụ na -ebelata ụkọ nke nitrogen na protein, na-emekwa ka nchịkọta nke glycogen nwee ike.

Nkwadebe isotonic nke 5% nwere ike jupụta ụkọ mmiri. Ọ nwere nsonaazụ na nsị metabolism, bụrụ onye na-eweta ihe oriri bara uru ma na-eme ngwa ngwa ngwa ngwa.

Site na ntinye nke glucose hypertonic 10%:

osmotic ọbara mgbali elu;
mmụba nke mmiri na-abanye n’ọbara;
a na-akpali usoro metabolic;
qualitative n’eme ka ọrụ nhicha ahụ dị ọcha;
diuresis na-abawanye.

Whomnye ka ọgwụ ahụ gosiri?

Usoro 5% nke a na-ahụ maka intravenly na-enye aka:

imeghari ngwa ngwa nke furu efu mmiri (na mkpokọta, extracellular na cellular akpọnwụ);
kpochapu onodu ujo na ida (dika otu n’ime ihe nke oke ujo ma oburu mmiri dochie anya obara).

Ihe ngwọta 10% nwere ụdị ngosipụta ndị a maka ojiji na nchịkwa intraven:

ya na akpịrị ịkpọ nkụ (ị na-agbọ ụfụ, na-agbakasị afọ, na oge ọrụ);
site na nsị na ụdị nsị ma ọ bụ ọgwụ ọjọọ (arsenic, ọgwụ ọjọọ, carbon monoxide, phosgene, cyanides, aniline);
ya na hypoglycemia, ịba ọcha n'anya, dystrophy, atrophy imeju, ọria ụbụrụ na ngụgụ, hemorrhagic diathesis, nsogbu nke obi na obi, ọrịa na-efe efe, ọrịa toxico;
n'oge nkwadebe nke ọgwụ ọgwụ maka nchịkwa ọbara (ịta 5% na 10%).

Kedụ ka m ga-esi eji ọgwụ ọjọọ eme ihe?

Ekwesịrị ịhapụ mmiri ọgwụ isotonic nke 5% na elekere kachasị elu nke 7 ml kwa nkeji (150 tụlee kwa nkeji ma ọ bụ 400 ml kwa awa).

Maka ndị okenye, ọgwụ ahụ nwere ike ịnye gị ọgwụ intravenously na oke 2 lita kwa ụbọchị. Ọ ga-ekwe omume ị theụ ọgwụ subcutaneously na enemas.

A na-egosipụta usoro hypertonic (10%) maka iji naanị site na nchịkwa intravenous na olu nke 20/40/50 ml kwa infusion. Ọ bụrụ na enwere ihe akaebe, wee kpọnye ya ọsọ ọsọ karịa 60 tụlee kwa nkeji. Usoro kachasị maka ndị okenye bụ 1000 ml.

O kwesiri ị ofụ ọgwụ ị intụ mmanya ga-adabere na mkpa nke otu ọ bụla. Ndị okenye na-enweghị oke ibu kwa ụbọchị nwere ike ịgaghị ihe karịrị 4-6 g / n'arọ kwa ụbọchị (ihe dịka 250-450 g kwa ụbọchị). N'okwu a, ọnụọgụ nke mmiri mmiri kwesịrị kwesịrị 30 ml / n'arọ kwa ụbọchị.

Site n'ibelata usoro metabolic dị ala, enwere akara iji belata ubochi kwa ụbọchị ruo 200-300 g.

Ọ bụrụ na achọrọ ọgwụgwọ ogologo oge, mgbe ahụ ekwesịrị ime nke a n'okpuru nyocha nke ọkwa shuga.

Maka nnabata glucose ngwa ngwa ma zuo ezu, n'ụzọ ụfọdụ, achọrọ insulin n'otu oge.

Mpempe nke mmeghachi omume na-adịghị mma maka ihe ahụ

Ntụziaka maka iji ya ekwuo na ihe mejupụtara ma ọ bụ ihe bụ isi n'ihe ụfọdụ nwere ike ibute mmeghachi omume anụ ahụ na nchịkwa glucose nke 10%, dịka ọmụmaatụ:

ahụ ọkụ
hypervolemia;
hyperglycemia;
nnukwu odida na ekpe ventricle.

Iji ogologo oge (ma ọ bụ site na ngwa ngwa oke nke nnukwu oke) nke ọgwụ nwere ike ime ka ọzịza, mmiri ịxụ mmiri, ọnọdụ imeju na-arụ ọrụ ma ọ bụ mwepu nke ngwa ikpuchi ume nke pancreas.

N'ebe ndị a nwere usoro njikọta akwara ọbara, mmepe nke ọrịa, thrombophlebitis na necrosis anụ ahụ ga-ekwe omume, na-aria ọbara ọgbụgba. Mmeghachi omume ndị ọzọ na nkwadebe glucose na ampoules nwere ike ịbụ nke ngwaahịa mepụtara ma ọ bụ jiri ụzọ nchịkwa na-ezighi ezi.

Site na nchịkwa intravenous, enwere ike ịchọpụta mmebi nke metabolism electrolyte:

hypokalemia;
hypophosphatemia;
hypomagnesemia.

Iji zere mmeghachi omume ọjọọ na mejupụtara ọgwụ ahụ na ndị ọrịa, ọ dị mkpa iji nlezianya debe usoro ị doụ ọgwụ a tụrụ aro ya na usoro nchịkwa kwesịrị ekwesị.

Isnye ka a na-egbochi glucose?

Ntuziaka maka iji nye ihe ọmụma banyere isi contraindications:

ọrịa shuga mellitus;
akụkụ ụbụrụ na akụkụ eriri afọ;
hyperglycemia;
hyperosmolar coma;
hyperlactacidemia;
mmebi akwara ọbara, na - eyi egwu mmepe nke ụbụrụ na ụbụrụ.

Mmekọrịta na ọgwụ ndị ọzọ

Ngwọta glucose nke 5% na 10% na ihe mejupụtara ya na-eme ka nnweta sodium dị mfe site na ngọngọ diges. Enwere ike ịkwado ọgwụ ahụ na ngwakọta ascorbic acid.

Nlekọta intravenous n’otu oge kwesịrị ịdị na ọnụego nke 1 unit kwa 4-5 g, nke na-enye aka na nha nke kacha arụ ọrụ.

N'iburu nke a, glucose 10% bụ ihe na - akpata oxidizing zuru oke nke enweghị ike ijikwa ya n'otu oge yana hexamethylenetetramine.

A na-ezere glucose kachasị mma na:

azịza alkaloids;
ọgwụ izugbe;
ọgwụ ịrahụ ụra.

Ngwọta ahụ nwere ike ịkụda nsonaazụ nke analgesics, ọgwụ adrenomimetic ma belata arụmọrụ nke nystatin.

Nufọdụ nuances

Mgbe ị na-a theụ ọgwụ ahụ, a ga-enyocha ọkwa shuga dị n'ọbara mgbe niile. Ntinye nke nnukwu glucose ike nwere ike bụrụ nke ndị ọrịa shuga na-enwe nnukwu elektrolyte. Enweghi ike iji ihe dochie 10% mgbe nnukwu ọgụ nke ischemia n'ụdị oke n'ihi mmetụta ọjọọ hyperglycemia na usoro ọgwụgwọ.